“Furthermore, while India has been polio-free for a year, there has been a huge increase in non-polio acute flaccid paralysis (NPAFP). In 2011, there were an extra 47,500 new cases of NPAFP. Clinically indistinguishable from polio paralysis but twice as deadly, the incidence of NPAFP was directly proportional to doses of oral polio received. Though this data was collected within the polio surveillance system, it was not investigated.”
http://www.ncbi.nlm.nih.gov/pubmed/22591873
Acute Fulminant Myocarditis after Diphtheria, Polio, and Tetanus Vaccination
“We report an infant case of acute fulminant myocarditis which occurred after administration of a diphtheria, polio, and tetanus vaccination. Fever and dyspnea developed after the vaccination. Extracorporeal membrane oxygenation was used for intractable cardiogenic shock. The patient survived the extracorporeal support, but poor ventricular contractility recurred 2 months later and she died while waiting for heart transplantation. ” “Hypersensitivity myocarditis due to vaccination has been reported sporadically.1–4 Occurrence of early fever after vaccination, negative results of microbial culture and the absence of associated symptoms for diphtheria, polio, and tetanus favor the diagnosis of hypersensitivity myocarditis. However, there was no definite evidence to support a causal link between the administration of vaccines and myocarditis. Repeated antigen injection is a well-established provocation test to substantiate a causal relationship, however this in itself raises ethical dilemmas.”
http://www.ncbi.nlm.nih.gov/pubmed/17130313
FULL TEXT HERE
Mayo Clinic Proc, Nov 2003
“Case.—A previously healthy 14-year-old boy presented with fever and intermittent (lasting a few minutes) chest pain. The symptoms developed 3 days after he had received a vaccination for tetanus (Tetavax, Aventis Pasteur SA, Lyon, France). His medical history was unremarkable except for a severe skin eruption that occurred after trimethoprim- sulfamethoxazole treatment when he was 8 years of age. He had no history of adverse reactions to tetanus or other vaccinations. One recent report described a case of myopericarditis after triple vaccination against diphtheria, tetanus, and poliovirus. The patient had symptoms similar to our patient’s, with slightly elevated cardiac enzymes and normal findings on echocardiography and coronary angiography. The vaccination was the suspected cause in view of the chronology of the symptoms. Performing a provocative test that would confirm the causal relationship between the vaccination and the cardiac anomalies would be unethical.”
“Hypersensitivity myocarditis should be considered when new ECG changes occur in association with acute-onset chest pain, mildly elevated cardiac enzyme levels, and eosinophilia due to drugs and vaccination.”
Mayo Clin Proc, November 2003, Vol 78
Acute Myopericarditis After Multiple Vaccinations in an Adolescent: Case Report and Review of the Literature
“Our case highlights the fact that pediatricians should be aware of the often-dramatic presentation of postvaccination myopericarditis and its usually benign clinical course. The diagnosis of myocarditis should be entertained when acute-onset chest pain is accompanied by ECG changes and elevated cardiac enzyme levels. In cases in which the above-described presentation is temporally related to routine immunizations, the immunizations should be considered as a possible underlying etiology. “
“The vaccination that has received great attention recently is that for smallpox, particularly after reinstitution of the vaccination for military personnel in 2002 and the reports of >50 cases of probable myocarditis temporally related to it”
http://pediatrics.aappublications.org/content/119/6/e1400.full
TWINS SHARE 100% OF THEIR DNA, THEY SHOULD HAVE A 100% AUTISM RATE IF IT WERE COMPLETELY GENETIC, BUT THEY DONT! AUTISM IS CAUSED BY ENVIRONMENTAL FACTORS.
Results For strict autism, probandwise concordance for male twins was 0.58 for 40 monozygotic pairs (95% confidence interval [CI], 0.42-0.74) and 0.21 for 31 dizygotic pairs (95% CI, 0.09-0.43); for female twins, the concordance was 0.60 for 7 monozygotic pairs (95% CI, 0.28-0.90) and 0.27 for 10 dizygotic pairs (95% CI, 0.09-0.69). For ASD, the probandwise concordance for male twins was 0.77 for 45 monozygotic pairs (95% CI, 0.65-0.86) and 0.31 for 45 dizygotic pairs (95% CI, 0.16-0.46); for female twins, the concordance was 0.50 for 9 monozygotic pairs (95% CI, 0.16-0.84) and 0.36 for 13 dizygotic pairs (95% CI, 0.11-0.60). A large proportion of the variance in liability can be explained by shared environmental factors (55%; 95% CI, 9%-81% for autism and 58%; 95% CI, 30%-80% for ASD) in addition to moderate genetic heritability (37%; 95% CI, 8%-84% for autism and 38%; 95% CI, 14%-67% for ASD).
Conclusion Susceptibility to ASD has moderate genetic heritability and a substantial shared twin environmental component.
http://www.ncbi.nlm.nih.gov/pubmed/21727249
FREE FULL TEXT STUDY HERE http://www.educate4theinjured.org/pdf-full-text-studies/4568262801
Neuro Endocrinol Lett. 2005 Oct;26(5):439-46.
Mercury and autism: accelerating evidence?
Autism spectrum disorders (ASDs) also known as pervasive developmental disorders (PDD) are a behaviorally defined group of neurodevelopmental disorders that are usually diagnosed in early childhood. ASDs disproportionately affect male children. Mercury (Hg) a heavy metal, is widespread and persistent in the environment. Mercury is a ubiquitous source of danger in fish, drugs, fungicides/herbicides, dental fillings, thermometers, and many other products.
Elevated Hg concentrations may remain in the brain from several years to decades following exposure. This is important because investigators have long recognized that Hg is a neurodevelopmental poison; it can cause problems in neuronal cell migration and division, and can ultimately cause cell degeneration and death. Case-reports of patients have described developmental regressions with ASD symptoms following fetal and/or early childhood Hg exposure, and epidemiological studies have linked exposure to Hg with an elevated risk of a patient being diagnosed with an ASD.
Immune, sensory, neurological, motor, and behavioral dysfunctions similar to traits defining or associated with ASDs were reported following Hg intoxication with similarities extending to neuroanatomy, neurotransmitters, and biochemistry.
The sexual dimorphism of ASDs may result from synergistic neurotoxicity caused by the interaction of testosterone and Hg; in contrast, estrogen is protective, mitigating the toxicity of Hg. Mercury exposure may significantly increase androgen levels, and as a result, patients diagnosed with an ASD may significantly benefit from anti-androgen therapy. Finally, the clinical geneticist has a wealth of biomarkers to evaluate and treat patients diagnosed with an ASD.
http://www.ncbi.nlm.nih.gov/pubmed/16264412
“Varicella vaccination is less effective than the natural immunity that existed in prevaccine communities. Universal varicella vaccination has not proven to be cost-effective as increased HZ morbidity has disproportionately offset cost savings associated with reductions in varicella disease. Universal varicella vaccination has failed to provide long-term protection from VZV disease.” PMID: 20642419
http://www.ncbi.nlm.nih.gov/pubmed/22659447
“Clustering of cases of insulin dependent diabetes (IDDM) occurring three years after hemophilus influenza B (HiB) immunization support causal relationship between immunization and IDDM (insulin depedent diabetes).”
http://www.ncbi.nlm.nih.gov/pubmed/12911277
“We initiated and funded a collaborative study with Tuomilehto on the effect of the Haemophilus influenzae type b vaccine on type 1 diabetes and found that the data support a causal relation (paper submitted for publication). Furthermore, the potential risk of the vaccine exceeds the potential benefit. We compared a group that received four doses of the vaccine, a group that received one dose, and a group that was not vaccinated. The cumulative incidence of diabetes per 100000 in the three groups receiving four, one, and no doses of the vaccine was 261, 237, and 207 at age 7 and 398, 376, and 340 at age 10 respectively.”
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1116914/
” The studies aiming to prove the widespread belief that healthcare worker vaccination decreases patient morbidity and mortality are heavily flawed and the recommendations for vaccination biased. No reliable published evidence shows that healthcare workers’ vaccination has substantial benefit for their patients—not in reducing patient morbidity or mortality and not even in increasing patient vaccination rates. Conclusion. The arguments for uniform healthcare worker influenza vaccination are not supported by existing literature. The decision whether to get vaccinated should, except possibly in extreme situations, be that of the individual healthcare worker, without legal, institutional, or peer coercion.”
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3502850/
Myocarditis after triple immunisation.
“We describe a 3 month old infant who developed myocarditis several hours after diphtheria, tetanus, and pertussis vaccination. The time of occurrence of symptoms, the clinical course, and the negative virological studies suggest a possible cardiogenic adverse reaction to the vaccine.
“Helle et al found electrocardiogram changes suggestive of myocarditis without evidence of cardiac disease in 3% of a study population consisting of new army recruits after vaccination against diphtheria and smallpox. In addition, several episodes of paroxysmal supraventricular tachycardia were observed within a few hours after diphtheria, tetanus, and pertussis immunisation in a 2 month old infant prone to paroxysmal supraventricular tachycardia.”
“We believe that the myocardial reaction described was associated with the diphtheria, tetanus, and pertussis vaccination. Of the three components of the vaccine, either the diphtheria or the pertussis
component probably provoked the myocardial damage.”
Myocarditis after Smallpox Vaccination: A Case Report
“A 20-year-old airman (US Air Force) developed myocarditis 8 days after smallpox vaccination. He was treated with nonsteroidal anti-inflammatory agents, and his symptoms promptly resolved. However, postvaccinial myocarditis can lead to serious complications and even death.”
“This patient clearly had myocarditis in temporal relation to primary smallpox vaccination. Viral and postviral myocarditis has been described in young servicemen, and therefore we tried aggressively to rule out nonvaccinial causes of the cardiac abnormalities in this patient, including common viral, autoimmune, bacterial, and toxicological etiologies. Several similar cases of postvaccinial myocarditis have recently been detected in US servicemen (J. Grabenstein, written communications, February and March 2003). Currently, the longterm prognosis of this condition is not known.”
“Clinicians providing care to patients who have chest complaints after smallpox vaccination should be aware of the existence of postvaccinial myocarditis, which seems to be more common than previously thought.”
http://cid.oxfordjournals.org/content/37/1/145.full.pdf+html
A case series of children with apparent mercury toxic encephalopathies manifesting with clinical symptoms of regressive autistic disorders.
Impairments in social relatedness and communication, repetitive behaviors, and stereotypic abnormal movement patterns characterize autism spectrum disorders (ASDs). It is clear that while genetic factors are important to the pathogenesis of ASDs, mercury exposure can induce immune, sensory, neurological, motor, and behavioral dysfunctions similar to traits defining or associated with ASDs. The Institutional Review Board of the Institute for Chronic Illnesses (Office for Human Research Protections, U.S. Department of Health and Human Services, IRB number IRB00005375) approved the present study. A case series of nine patients who presented to the Genetic Centers of America for a genetic/developmental evaluation are discussed. Eight of nine patients (one patient was found to have an ASD due to Rett’s syndrome) (a) had regressive ASDs; (b) had elevated levels of androgens; (c) excreted significant amounts of mercury post chelation challenge; (d) had biochemical evidence of decreased function in their glutathione pathways; (e) had no known significant mercury exposure except from Thimerosal-containing vaccines/Rho(D)-immune globulin preparations; and (f) had alternate causes for their regressive ASDs ruled out. There was a significant dose-response relationship between the severity of the regressive ASDs observed and the total mercury dose children received from Thimerosal-containing vaccines/Rho (D)-immune globulin preparations. Based upon differential diagnoses, 8 of 9 patients examined were exposed to significant mercury from Thimerosal-containing biologic/vaccine preparations during their fetal/infant developmental periods, and subsequently, between 12 and 24 mo of age, these previously normally developing children suffered mercury toxic encephalopathies that manifested with clinical symptoms consistent with regressive ASDs. Evidence for mercury intoxication should be considered in the differential diagnosis as contributing to some regressive ASDs.
http://www.ncbi.nlm.nih.gov/pubmed/17454560
Autism: a novel form of mercury poisoning
“Thimerosal, a preservative added to many vaccines, has become a major source of mercury in children who, within their first two years, may have received a quantity of mercury that exceeds safety guidelines. A review of medical literature and US government data suggests that: (i) many cases of idiopathic autism are induced by early mercury exposure from thimerosal; (ii) this type of autism represents an unrecognized mercurial syndrome; and (iii) genetic and non-genetic factors establish a predisposition whereby thimerosal’s adverse effects occur only in some children.”
http://www.ncbi.nlm.nih.gov/pubmed/11339848
Annual influenza vaccination affects the development of heterosubtypic immunity. 2012 May 27.
Annual vaccination of healthy children >6 months of age against seasonal influenza has been recommended by public health authorities of some countries. However, currently used seasonal vaccines provide only limited protection against (potentially) pandemic influenza viruses. Furthermore, we recently hypothesized that annual vaccination may hamper the development of cross-reactive immunity against influenza A viruses of novel subtypes, that would otherwise be induced by natural infection. Here we summarize our findings in animal models in which we demonstrated that vaccination against influenza A/H3N2 virus reduced the induction of heterosubtypic immunity against highly pathogenic avian influenza A/H5N1 virus, otherwise induced by a prior infection with influenza A/H3N2 virus. The reduction of heterosubtypic immunity correlated with reduced virus-specific CD8+ T cell responses. An additional study was performed in humans, in which we collected peripheral blood mononuclear cells from annually vaccinated children with cystic fibrosis (CF) and age-matched unvaccinated healthy control children to study the virus-specific T cell response. An age-related increase of the virus-specific CD8+ T cell response was observed in unvaccinated children that was absent in vaccinated children with CF. These findings highlight the importance of the development of vaccines that provide protection against influenza A viruses of all subtypes. PMID: 22643217
http://www.ncbi.nlm.nih.gov/pubmed/22643217
“Together with an inflammatory reaction, influenza A vaccine induced platelet activation and sympathovagal imbalance towards adrenergic predominance. Significant correlations were found between CRP levels and HRV parameters, suggesting a pathophysiological link between inflammation and cardiac autonomic regulation. The vaccine-related platelet activation and cardiac autonomic dysfunction may transiently increase the risk of cardiovascular events.”
http://www.ncbi.nlm.nih.gov/pubmed/20964738
“During 2009-2010 they found that the risk of narcolepsy among people aged 4-19 years old who had received pandemic influenza vaccine was nine times higher than that among those who had not been vaccinated.”
All Medical & Health Advice should be decided with a competent Medical Professional. This site does not guarantee anyones health or claim to be a medical website. We encourage education before vaccination.
“The US childhood immunization schedule requires 26 vaccine doses for infants aged less than 1 year, THE MOST IN THE WORLD, yet 33 nations have better Infant Mortality Rates (IMR). Using linear regression, the immunization schedules of these 34 nations were examined and a correlation coefficient of 0.70 (p < 0.0001) was found between IMRs and the number of vaccine doses routinely given to infants. When nations were grouped into five different vaccine dose ranges (12–14, 15–17, 18–20, 21–23, and 24–26), 98.3% of the total variance in IMR was explained by the unweighted linear regression model.
These findings demonstrate a counter-intuitive relationship:
nations that require more vaccine doses tend to have higher infant mortality rates.
Efforts to reduce the relatively high UNITED STATES INFANT MORTALITY RATE have been elusive. Finding ways to lower preterm birth rates should be a high priority. However, preventing premature births is just a partial solution to reduce infant deaths. A closer inspection of correlations between vaccine doses, biochemical or synergistic toxicity, and IMRs, is essential. All nations—rich and poor, advanced and developing—have an obligation to determine whether their immunization schedules are achieving their desired goals.”
This scientific study can be seen here with all models:
http://het.sagepub.com/content/early/2011/05/04/0960327111407644.full.pdf+htm
Adverse Effects of Pertussis and Rubella Vaccines (1991)
A Report of the Committee to Review the Adverse Consequences of Pertussis and Rubella Vaccines
https://www.ncbi.nlm.nih.gov/books/NBK234363/
https://www.ncbi.nlm.nih.gov/books/NBK234363/pdf/Bookshelf_NBK234363.pdf
World Mercury Project – Robert F. Kennedy Jr.
